Fisiología
Departamento
Harvard Medical School
Boston, Estados UnidosPublicaciones en colaboración con investigadoras/es de Harvard Medical School (9)
2016
2013
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Acetylation and deacetylation - Novel factors in muscle wasting
Metabolism: Clinical and Experimental, Vol. 62, Núm. 1, pp. 1-11
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CaMKII activity is reduced in skeletal muscle during sepsis
Journal of Cellular Biochemistry, Vol. 114, Núm. 6, pp. 1294-1305
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PPARβ/δ Regulates Glucocorticoid- and Sepsis-Induced FOXO1 Activation and Muscle Wasting
PLoS ONE, Vol. 8, Núm. 3
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Suppression of atrogin-1 and MuRF1 prevents dexamethasone-induced atrophy of cultured myotubes
Metabolism: Clinical and Experimental, Vol. 62, Núm. 10, pp. 1495-1502
2012
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Loss of muscle strength during sepsis is in part regulated by glucocorticoids and is associated with reduced muscle fiber stiffness
American Journal of Physiology - Regulatory Integrative and Comparative Physiology, Vol. 303, Núm. 10
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Resveratrol prevents dexamethasone-induced expression of the muscle atrophy-related ubiquitin ligases atrogin-1 and MuRF1 in cultured myotubes through a SIRT1-dependent mechanism
Biochemical and Biophysical Research Communications, Vol. 417, Núm. 1, pp. 528-533
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β-Hydroxy-β-methylbutyrate (HMB) prevents dexamethasone-induced myotube atrophy
Biochemical and Biophysical Research Communications, Vol. 423, Núm. 4, pp. 739-743