Biología Celular e Histología
Departamento
Icahn School of Medicine at Mount Sinai
Nueva York, Estados UnidosPublicaciones en colaboración con investigadoras/es de Icahn School of Medicine at Mount Sinai (11)
2017
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Regulation of m6A Transcripts by the 3ʹ→5ʹ RNA Helicase YTHDC2 Is Essential for a Successful Meiotic Program in the Mammalian Germline
Molecular Cell, Vol. 68, Núm. 2, pp. 374-387.e12
2014
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Extracellular matrix and liver disease
Antioxidants and Redox Signaling, Vol. 21, Núm. 7, pp. 1078-1097
2012
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Discoidin domain receptor 2 deficiency predisposes hepatic tissue to colon carcinoma metastasis
Gut, Vol. 61, Núm. 10, pp. 1465-1472
2011
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Impaired dermal wound healing in discoidin domain receptor 2-deficient mice associated with defective extracellular matrix remodeling
Fibrogenesis and Tissue Repair, Vol. 4, Núm. 1
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Loss of discoidin domain receptor 2 promotes hepatic fibrosis after chronic carbon tetrachloride through altered paracrine interactions between hepatic stellate cells and liver-associated macrophages
American Journal of Pathology, Vol. 179, Núm. 6, pp. 2894-2904
2003
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Proangiogenic role of tumor-activated hepatic stellate cells in experimental melanoma metastasis
Hepatology, Vol. 37, Núm. 3, pp. 674-685
2002
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Discoidin domain receptor 2 interacts with Src and Shc following its activation by type I collagen
Journal of Biological Chemistry, Vol. 277, Núm. 21, pp. 19206-19212
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Discoidin domain receptor 2 regulates fibroblast proliferation and migration through the extracellular matrix in association with transcriptional activation of matrix metalloproteinase-2
Journal of Biological Chemistry, Vol. 277, Núm. 5, pp. 3606-3613
2001
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DDR2 receptor promotes MMP-2-mediated proliferation and invasion by hepatic stellate cells
Journal of Clinical Investigation, Vol. 108, Núm. 9, pp. 1369-1378
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The collagen receptor DDR2 regulates proliferation and its elimination leads to dwarfism
EMBO Reports, Vol. 2, Núm. 5, pp. 446-452
1998
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Molecular regulation of hepatic fibrogenesis
Journal of Hepatology, Vol. 29, Núm. 5, pp. 836-847