Publicaciones en las que colabora con Juan Anguita (13)

2023

  1. Mitochondrial dysfunction promotes microbial composition that negatively impacts on ulcerative colitis development and progression

    npj Biofilms and Microbiomes, Vol. 9, Núm. 1

2022

  1. Mitochondrial bioenergetics boost macrophage activation, promoting liver regeneration in metabolically compromised animals

    Hepatology, Vol. 75, Núm. 3, pp. 550-566

  2. Restoring cellular magnesium balance through Cyclin M4 protects against acetaminophen-induced liver damage

    Nature Communications, Vol. 13, Núm. 1

2021

  1. Borrelia burgdorferi infection induces long-term memory-like responses in macrophages with tissue-wide consequences in the heart

    PLoS Biology, Vol. 19, Núm. 1 December

2019

  1. Regulation of macrophage activity by surface receptors contained within Borrelia burgdorferi-enriched phagosomal fractions

    PLoS Pathogens, Vol. 15, Núm. 11

  2. The Mycobacterium tuberculosis capsule: A cell structure with key implications in pathogenesis

    Biochemical Journal, Vol. 476, Núm. 14, pp. 1995-2016

  3. miR-873-5p targets mitochondrial GNMT-Complex II interface contributing to non-alcoholic fatty liver disease

    Molecular Metabolism, Vol. 29, pp. 40-54

2018

  1. A multi-omic analysis reveals the regulatory role of CD180 during the response of macrophages to Borrelia burgdorferi article

    Emerging Microbes and Infections, Vol. 7, Núm. 1

2017

  1. MTORC1-dependent AMD1 regulation sustains polyamine metabolism in prostate cancer

    Nature, Vol. 547, Núm. 7661, pp. 109-113

  2. Role of aramchol in steatohepatitis and fibrosis in mice

    Hepatology Communications, Vol. 1, Núm. 9, pp. 911-927

  3. The immunosuppressive effect of the tick protein, Salp15, is long-lasting and persists in a murine model of hematopoietic transplant

    Scientific Reports, Vol. 7, Núm. 1

2016

  1. Stratification and therapeutic potential of PML in metastatic breast cancer

    Nature Communications, Vol. 7

2015

  1. Ikaros mediates the DNA methylation-independent silencing of MCJ/DNAJC15 gene expression in macrophages

    Scientific Reports, Vol. 5