Publicaciones en colaboración con investigadoras/es de Novartis (Sweden) (6)

2012

  1. Dispersible amyloid β-protein oligomers, protofibrils, and fibrils represent diffusible but not soluble aggregates: Their role in neurodegeneration in amyloid precursor protein (APP) transgenic mice

    Neurobiology of Aging, Vol. 33, Núm. 11, pp. 2641-2660

  2. Transgenic expression of intraneuronal Aβ42 but not Aβ40 leads to cellular Aβ lesions, degeneration, and functional impairment without typical Alzheimer's disease pathology

    Journal of Neuroscience, Vol. 32, Núm. 4, pp. 1273-1283

2009

  1. Capillary cerebral amyloid angiopathy is associated with vessel occlusion and cerebral blood flow disturbances

    Neurobiology of Aging, Vol. 30, Núm. 12, pp. 1936-1948

2007

  1. Occurrence and co-localization of amyloid β-protein and apolipoprotein E in perivascular drainage channels of wild-type and APP-transgenic mice

    Neurobiology of Aging, Vol. 28, Núm. 8, pp. 1221-1230

2006

  1. Selective vulnerability of different types of commissural neurons for amyloid β-protein-induced neurodegeneration in APP23 mice correlates with dendritic tree morphology

    Brain, Vol. 129, Núm. 11, pp. 2992-3005

1998

  1. The multidrug resistance modulator valspodar (PSC 833) is metabolized by human cytochrome P450 3A: Implications for drug-drug interactions and pharmacological activity of the main metabolite

    Drug Metabolism and Disposition, Vol. 26, Núm. 8, pp. 802-811