Publicaciones en colaboración con investigadoras/es de University of Bonn (7)

2012

  1. Dispersible amyloid β-protein oligomers, protofibrils, and fibrils represent diffusible but not soluble aggregates: Their role in neurodegeneration in amyloid precursor protein (APP) transgenic mice

    Neurobiology of Aging, Vol. 33, Núm. 11, pp. 2641-2660

2009

  1. Capillary cerebral amyloid angiopathy is associated with vessel occlusion and cerebral blood flow disturbances

    Neurobiology of Aging, Vol. 30, Núm. 12, pp. 1936-1948

2008

  1. Inter-laboratory comparison of neuropathological assessments of β-amyloid protein: A study of the BrainNet Europe consortium

    Acta Neuropathologica, Vol. 115, Núm. 5, pp. 533-546

  2. Tracing of temporo-entorhinal connections in the human brain: Cognitively impaired argyrophilic grain disease cases show dendritic alterations but no axonal disconnection of temporo-entorhinal association neurons

    Acta Neuropathologica, Vol. 115, Núm. 2, pp. 175-183

2006

  1. Selective vulnerability of different types of commissural neurons for amyloid β-protein-induced neurodegeneration in APP23 mice correlates with dendritic tree morphology

    Brain, Vol. 129, Núm. 11, pp. 2992-3005

  2. The development of amyloid beta protein deposits in the aged brain.

    Science of aging knowledge environment : SAGE KE, Vol. 2006, Núm. 6

2005

  1. Apolipoprotein E co-localizes with newly formed amyloid β-protein (Aβ) deposits lacking immunoreactivity against N-terminal epitopes of Aβ in a genotype-dependent manner

    Acta Neuropathologica, Vol. 110, Núm. 5, pp. 459-471