University of Milan-ko ikertzaileekin lankidetzan egindako argitalpenak (11)

2023

  1. Stimulation of synaptic activity promotes TFEB-mediated clearance of pathological MAPT/Tau in cellular and mouse models of tauopathies

    Autophagy, Vol. 19, Núm. 2, pp. 660-677

2019

  1. Exome sequencing in multiple sclerosis families identifies 12 candidate genes and nominates biological pathways for the genesis of disease

    PLoS Genetics, Vol. 15, Núm. 6

2018

  1. Synaptic activity protects against AD and FTD-like pathology via autophagic-lysosomal degradation

    Molecular Psychiatry, Vol. 23, Núm. 6, pp. 1530-1540

2008

  1. Refining genetic associations in multiple sclerosis

    The Lancet Neurology

2006

  1. P2Y1 receptor-evoked glutamate exocytosis from astrocytes: Control by tumor necrosis factor-α and prostaglandins

    Journal of Biological Chemistry, Vol. 281, Núm. 41, pp. 30684-30696

2001

  1. CXCR4-activated astrocyte glutamate release via TNFa: Amplification by microglia triggers neurotoxicity

    Nature Neuroscience, Vol. 4, Núm. 7, pp. 702-710

  2. Neuron-astrocyte cross-talk during synaptic transmission: Physiological and neuropathological implications

    Progress in Brain Research, Vol. 132, pp. 255-265

  3. The expression of glutamate transporter GLT-1 in the rat cerebral cortex is down-regulated by the antipsychotic drug clozapine

    Molecular Psychiatry, Vol. 6, Núm. 4, pp. 380-386

2000

  1. Glial cells couple inflammation to glutamate neurodegeration: Let's cut it there

    Neurological Sciences, Vol. 21, Núm. 4 SUPPL.

1997

  1. Occurrence and clinical relevance of an interleukin-4 gene polymorphism in patients with multiple sclerosis

    Journal of Neuroimmunology, Vol. 76, Núm. 1-2, pp. 189-192

  2. Tumor necrosis factor α and its receptors in relapsing-remitting multiple sclerosis

    Journal of the Neurological Sciences, Vol. 152, Núm. 1, pp. 51-61