Laboratory of Neurogenomiks
Novartis (Sweden)
Täby, SueciaNovartis (Sweden)-ko ikertzaileekin lankidetzan egindako argitalpenak (5)
2012
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Dispersible amyloid β-protein oligomers, protofibrils, and fibrils represent diffusible but not soluble aggregates: Their role in neurodegeneration in amyloid precursor protein (APP) transgenic mice
Neurobiology of Aging, Vol. 33, Núm. 11, pp. 2641-2660
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Transgenic expression of intraneuronal Aβ42 but not Aβ40 leads to cellular Aβ lesions, degeneration, and functional impairment without typical Alzheimer's disease pathology
Journal of Neuroscience, Vol. 32, Núm. 4, pp. 1273-1283
2009
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Capillary cerebral amyloid angiopathy is associated with vessel occlusion and cerebral blood flow disturbances
Neurobiology of Aging, Vol. 30, Núm. 12, pp. 1936-1948
2007
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Occurrence and co-localization of amyloid β-protein and apolipoprotein E in perivascular drainage channels of wild-type and APP-transgenic mice
Neurobiology of Aging, Vol. 28, Núm. 8, pp. 1221-1230