Araba
Campus
Gunma University
Maebashi, JapónPublications en collaboration avec des chercheurs de Gunma University (5)
2012
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Dispersible amyloid β-protein oligomers, protofibrils, and fibrils represent diffusible but not soluble aggregates: Their role in neurodegeneration in amyloid precursor protein (APP) transgenic mice
Neurobiology of Aging, Vol. 33, Núm. 11, pp. 2641-2660
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Transgenic expression of intraneuronal Aβ42 but not Aβ40 leads to cellular Aβ lesions, degeneration, and functional impairment without typical Alzheimer's disease pathology
Journal of Neuroscience, Vol. 32, Núm. 4, pp. 1273-1283
2007
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Occurrence and co-localization of amyloid β-protein and apolipoprotein E in perivascular drainage channels of wild-type and APP-transgenic mice
Neurobiology of Aging, Vol. 28, Núm. 8, pp. 1221-1230
2006
2005
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Apolipoprotein E co-localizes with newly formed amyloid β-protein (Aβ) deposits lacking immunoreactivity against N-terminal epitopes of Aβ in a genotype-dependent manner
Acta Neuropathologica, Vol. 110, Núm. 5, pp. 459-471