Publications dans lesquelles il/elle collabore avec Rosa Barrio (14)

2021

  1. Identification of proximal SUMO-dependent interactors using SUMO-ID

    Nature Communications, Vol. 12, Núm. 1

2020

  1. Genetic manipulation of LKB1 elicits lethal metastatic prostate cancer

    The Journal of experimental medicine, Vol. 217, Núm. 6

  2. HuR/ELAVL1 drives malignant peripheral nerve sheath tumor growth and metastasis

    The Journal of clinical investigation, Vol. 130, Núm. 7, pp. 3848-3864

  3. LUZP1, a novel regulator of primary cilia and the actin cytoskeleton, is a contributing factor in townes-brocks syndrome

    eLife, Vol. 9, pp. 1-84

  4. Targeting PML in triple negative breast cancer elicits growth suppression and senescence

    Cell Death and Differentiation, Vol. 27, Núm. 4, pp. 1186-1199

2018

  1. Integrative analysis of transcriptomics and clinical data uncovers the tumor-suppressive activity of MITF in prostate cancer

    Cell Death and Disease, Vol. 9, Núm. 10

  2. PPARd elicits ligand-independent repression of trefoil factor family to limit prostate cancer growth

    Cancer Research, Vol. 78, Núm. 2, pp. 399-409

2017

  1. MTORC1-dependent AMD1 regulation sustains polyamine metabolism in prostate cancer

    Nature, Vol. 547, Núm. 7661, pp. 109-113

  2. The immunosuppressive effect of the tick protein, Salp15, is long-lasting and persists in a murine model of hematopoietic transplant

    Scientific Reports, Vol. 7, Núm. 1

2016

  1. Stratification and therapeutic potential of PML in metastatic breast cancer

    Nature Communications, Vol. 7

  2. The metabolic co-regulator PGC1α suppresses prostate cancer metastasis

    Nature Cell Biology, Vol. 18, Núm. 6, pp. 645-656

  3. Transcriptomic profiling of urine extracellular vesicles reveals alterations of CDH3 in prostate cancer

    Oncotarget, Vol. 7, Núm. 6, pp. 6835-6846

2015

  1. Ikaros mediates the DNA methylation-independent silencing of MCJ/DNAJC15 gene expression in macrophages

    Scientific Reports, Vol. 5

2014

  1. Tetramerization-defects of p53 result in aberrant ubiquitylation and transcriptional activity

    Molecular Oncology, Vol. 8, Núm. 5, pp. 1026-1042