CARLOS JOSE
MATUTE ALMAU
PROFESORADO CATEDRATICO/A DE UNIVERSIDAD
ASIER
RUIZ NUÑEZ
PROFESORADO AGREGADO
Publikationen, an denen er mitarbeitet ASIER RUIZ NUÑEZ (16)
2023
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Hepatic levels of S-adenosylmethionine regulate the adaptive response to fasting
Cell Metabolism, Vol. 35, Núm. 8, pp. 1373-1389.e8
2022
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Amyloid β / PKC-dependent alterations in NMDA receptor composition are detected in early stages of Alzheimer´s disease
Cell Death and Disease, Vol. 13, Núm. 3
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GABA Receptor Agonists Protect From Excitotoxic Damage Induced by AMPA in Oligodendrocytes
Frontiers in Pharmacology, Vol. 13
2021
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Role of Mitochondrial Dynamics in Microglial Activation and Metabolic Switch
ImmunoHorizons, Vol. 5, Núm. 8, pp. 615-626
2020
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Mitochondrial division inhibitor 1 disrupts oligodendrocyte Ca2+ homeostasis and mitochondrial function
GLIA, Vol. 68, Núm. 9, pp. 1743-1756
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Sephin1 protects neurons against excitotoxicity independently of the integrated stress response
International Journal of Molecular Sciences, Vol. 21, Núm. 17, pp. 1-11
2018
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Blockade and knock-out of CALHM1 channels attenuate ischemic brain damage
Journal of Cerebral Blood Flow and Metabolism, Vol. 38, Núm. 6, pp. 1060-1069
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Inhibition of Casein Kinase 2 Protects Oligodendrocytes From Excitotoxicity by Attenuating JNK/p53 Signaling Cascade
Frontiers in Molecular Neuroscience, Vol. 11
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Mangiferin and morin attenuate oxidative stress, mitochondrial dysfunction, and neurocytotoxicity, induced by Amyloid Beta Oligomers
Oxidative Medicine and Cellular Longevity, Vol. 2018
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Mitochondrial division inhibitor 1 (Mdivi-1) protects neurons against excitotoxicity through the modulation of mitochondrial function and intracellular Ca2+ signaling
Frontiers in Molecular Neuroscience, Vol. 11
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Re-examining the potential of targeting ABHD6 in multiple sclerosis: Efficacy of systemic and peripherally restricted inhibitors in experimental autoimmune encephalomyelitis
Neuropharmacology, Vol. 141, pp. 181-191
2014
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CGP37157, an inhibitor of the mitochondrial Na+/Ca2+ exchanger, protects neurons from excitotoxicity by blocking voltage-gated Ca2+ channels
Cell Death and Disease, Vol. 5, Núm. 4
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Calcium dyshomeostasis in white matter injury
White Matter Injury in Stroke and CNS Disease (Springer New York), pp. 433-460
2013
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1–42 β-amyloid peptide requires PDK1/nPKC/Rac 1 pathway to induce neuronal death
Translational Psychiatry, Vol. 3, Núm. 1
2010
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Intracellular Ca2+ release through ryanodine receptors contributes to AMPA receptor-mediated mitochondrial dysfunction and ER stress in oligodendrocytes
Cell Death and Disease, Vol. 1, Núm. 7
2009
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Endoplasmic reticulum Ca2+ release through ryanodine and IP3 receptors contributes to neuronal excitotoxicity
Cell Calcium, Vol. 46, Núm. 4, pp. 273-281