Bases moleculares de patologías humanas de alta prevalencia: cáncer e infertilidad. Avanzando (en medicina de precisión) hacia un manejo personalizado del paciente - GIC 21/046
Ikerbasque, Fundación Vasca para la Ciencia
Bilbao, EspañaPublications en collaboration avec des chercheurs de Ikerbasque, Fundación Vasca para la Ciencia (12)
2023
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Foreword Special Issue Genomic Instability in Tumor Evolution and Therapy Response
Cancers
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Mitochondrial dysfunction promotes microbial composition that negatively impacts on ulcerative colitis development and progression
npj Biofilms and Microbiomes, Vol. 9, Núm. 1
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Mitochondrial dysfunction-associated microbiota establishes a transmissible refractory response to anti-TNF therapy during ulcerative colitis
Gut Microbes, Vol. 15, Núm. 2
2022
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E2f2 attenuates apoptosis of activated t lymphocytes and protects from immune‐mediated injury through repression of fas and fasl
International Journal of Molecular Sciences, Vol. 23, Núm. 1
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Mitochondrial complex I dysfunction alters the balance of soluble and membrane-bound TNF during chronic experimental colitis
Scientific Reports, Vol. 12, Núm. 1
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Targeting E2F Sensitizes Prostate Cancer Cells to Drug-Induced Replication Stress by Promoting Unscheduled CDK1 Activity
Cancers, Vol. 14, Núm. 19
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microRNA-based signatures obtained from endometrial fluid identify implantative endometrium
Human Reproduction, Vol. 37, Núm. 10, pp. 2375-2391
2021
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A proteomic approach for systematic mapping of substrates of human deubiquitinating enzymes
International Journal of Molecular Sciences, Vol. 22, Núm. 9
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E2F1 and E2F2-mediated repression of CPT2 establishes a lipid-rich tumor-promoting environment
Cancer Research, Vol. 81, Núm. 11, pp. 2874-2887
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LUZP1 Controls Cell Division, Migration and Invasion Through Regulation of the Actin Cytoskeleton
Frontiers in Cell and Developmental Biology, Vol. 9
2020
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The mitochondrial negative regulator MCJ modulates the interplay between microbiota and the host during ulcerative colitis
Scientific Reports, Vol. 10, Núm. 1
2012
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Rac1 protein regulates glycogen phosphorylase activation and controls interleukin (IL)-2-dependent T cell proliferation
Journal of Biological Chemistry, Vol. 287, Núm. 15, pp. 11878-11890